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To estimate the correlation between changes in the expression of (h)GSH biosynthesis-related genes and actual thiol levels, a quantitative determination of low-molecular-weight thiols in 1-, 2-, and 3-week-old roots and nodules was undertaken (Figures 6A,B)

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Studies suggest that KPVs anti-inflammatory mechanism is independent of melanocortin receptors and involves direct entry into cells, distinguishing it from full-length -MSH signaling

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Conclusion: Our findings indicate that the liver microenvironment in MASLD impairs the ability of Tregs to suppress effector immune cell activation, thus perpetuating chronic inflammation and driving MASLD progression

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